Chemical Matrixectomy by Stacy Witfill DPM

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Onychocryptosis or more commonly referred to or known as ingrown nails can be painful and dangerous for high risk diabetic patients and patients with peripheral vascular disease. The condition can be painful for any patient and often “even the bed sheets cause the toe to hurt” The following article outlines a permanent procedure for removing ingrown toenails. Diabetic patient should first be assessed for peripheral vascular disease and glycemic control before performing the procedure. Consideration should be made to the kidney function of patients as 5-10 percent of these patients will develop post-operative infections and require systemic antibiotics. These considerations should be discussed with the patient during the pre-operative visit.

In addition care should be taken to fully evaluate the patient for any associated benign or malignant tumors involving the nail before performing a chemical matrixectomy. Although rare, any unusual “proud flesh” or unusual pigmentation of the nail bed should be biopsied before performing a chemical matrixectomy. Such benign tumors include warts(periungual and subungual), fibroma, neurofibroma, pigmented nevus, pyogenic granuloma(periungual and subungual), glomus tumor,myxoid cyst, acquired digital fibrokeratoma,keratoacanthoma, bone cysts including subungual exostosis,osteochondroma,enchondroma. Malignant tumors include bowens disease,metastatic carcinoma most commonly associated with a club appearing nail, basal cell carcinoma,malignant melanoma, and squamous cell carcinoma(the most common malignant cancer see associated with ingrown nails. If there is any clinical suspicious findings which could indicate any of the above conditions the patient should be advised of this and appropriate biopsy carried out.

There are also associated biomechanical etiologies of ingrown toenails that can be considered before performing a chemical matrixectomy. For example hallux abducto valgus deformity that is severe enough to cause the lateral border of the nail to impinge against the medial border of the 2nd toe causing the nail fold to overgrow the nail plate and thus causing the nail margin to embed within the tissue. In this situation the patient should be at least advised on the underlying etiology and at least given the option to trying a simple pad between the toes as an alternative to a chemical matrixectomy.

Performing a chemical matrixectomy

Infiltrate 3 cc of lidocaine 2% in standard hallux block. Standard prep with iodine. Introduce a freer elevator under the hyponychium and the eponychium Using a longitudinal nail splitter, create a vertical incision into the nail plate the level of the proximal nail plate. Utilize a 61 blade to finish the cut under the eponychium. Remove the offending nail border with a straight hemostat. Apply 89% phenol for 30 seconds. Repeat 2 applications of phenol. Rinse copiously with alcohol. Utilize a currette and remove excess tissue. Apply silvadene with a dry sterile compressive dressing. Follow patient as needed every 1-2 weeks.

 

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Winter Sunrise by Stacy Witfill

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Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

Stacy Witfill

Winter Sunrise by
Stacy Witfill

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Ocean by Stacy Witfill

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Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

Stacy Witfill

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Stacy Witfill | about.me

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Hammer Toes | Foot Health | Learn About Feet | APMA

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Hammer Toes | Foot Health | Learn About Feet | APMA.

Neuropathy

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Peripheral Neuropathy results from damage to the peripheral nervous system, which is responsible for transmitting information between the brain, the spinal cord, and the rest of the body. Hundreds of types of peripheral neuropathy have been identified, each with a defining set of symptoms. People commonly report numbness, tingling, and a prickly sensations. Others develop an extreme sensitivity to touch, while others develop muscle weakness and muscle wasting. Severe symptoms can include burning pain which is typically worse at night, limb paralysis, and organ or gland dysfunction.

Autonomic Neuropathy is a group of disorders that damage the nerves supplying the internal body structures that regulate numerous ‘automatic’ body functions such as blood pressure, heart rate, sweating, and bowel, bladder, and sexual function. If the cause can be identified and treated, the autonomic nerves may repair or regenerate.

Symptoms may improve with proper treatment. Most symptoms of autonomic neuropathy are uncomfortable but they can be debilitating in severe cases, especially if adequate blood pressure is not maintained while standing (this is called orthostatic hypotension).

There is a large variety of treatment options available for peripheral neuropathy. Some are more successful than others and some are dependent on the causes and type of neuropathy being treated.

Diagnosing Neuropathy can be difficult and can not always be diagnosed by clinical history and exam alone.
Traditionally EMG and NCV studies are utilized to confirm neuropathy, however in the early stages of small fiber neuropathy the most common form of neuropathy that diabetic patients have the NCV studies are often normal.Â

At Dunnellon Podiatry Center, Dr. Witfill often utilizes epidermal nerve fiber density testing to diagnose small fiber neuropathy.
Epidermal nerve fiber density testing (ENFD) is not a new technology, in fact, this technique has been used by neurologists for roughly 15 years. This test takes advantage of the fact that most forms of peripheral neuropathy progress in a distal to proximal fashion, beginning with the body smallest and most distal nerve twigs (C fibers and A delta fibers), and then progressing proximally.

This is why our diabetic patients so often present with peripheral neuropathy in a stocking like distribution. Those that exhibit neuropathy in this pattern, without involvement of large nerve fibers are said to have small fiber peripheral neuropathy. If the nerve pathology progresses proximally to involve larger nerves, the neuropathy becomes mixed.